Inflammation of nervous-system tissue, a target in many neuro protocols.
Chronic, low-level immune activation in the brain and spinal cord—distinct from acute infection—implicated in Parkinson's, Alzheimer's, and multiple sclerosis. Stem cells are studied for neuroinflammation because they release anti-inflammatory signals that cross the blood-brain barrier. Clinics treating neurological conditions often invoke neuroinflammation, which is scientifically sound, but brain delivery is mechanically challenging; cells injected intravenously or intrathecally may not reach all inflamed regions. Ask clinics how they ensure distribution and whether they monitor neuroinflammatory markers (glial activation, cytokine panels) before and after treatment to prove the mechanism.
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